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Macrophage Mal1 deficiency suppresses atherosclerosis in low-density lipoprotein receptor-null mice by activating peroxisome proliferator-activated receptor-g-regulated genes

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Academic year: 2021

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Figure 4. Treatment with palmitic acid complexed with BSA (A and B) or lipopolysaccharide (C to F) significantly increased activity of Akt and slightly suppressed NF- ␬B-related protein and genes in Mal1-deficient macrophages
Figure I. Expression of PPARγ-related genes in WT and Mal1-/- peritoneal macrophages treated  with AcLDL or OxLDL for 48 hours.
Figure III. Identification of a putative PPARγ responsive element (PPRE) in the mouse and  human PPAR-gamma genes.

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