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Pulmonary valve endocarditis Pulmoner kapak endokarditi

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Pulmonary valve endocarditis

Pulmoner kapak endokarditi

Tekin Y›ld›r›m, MD, C.Selim ‹flbir, MD*, Sinan Arsan, MD*

Department of Cardiovascular Surgery, Göztepe fiafak Hospital

*Department of Cardiovascular Surgery, Marmara University School of Medicine, ‹stanbul, Turkey

An eleven years old boy was admitted to another hospital with fever, malaise and fatigue. On physical examination 3/6 systolic murmur in mesocardium was noticed and echocardiog-raphy was performed which showed a ventricular septal defect (VSD) with a vegetation over pulmonary, valve and pulmonary artery pressure of 90 mmHg. The patient’s family was unaware of the VSD previously. The patient was hospitalized with the di-agnosis of subacute endocarditis and triple antibiotic regimen was started. The blood cultures were negative during this time despite the fever. The echocardiography was repeated and a large outlet VSD with pulmonary valve gradient of 50 mmHg and

vegetation over the pulmonary valve were reported. Meanwhile patient had several small infarcts in his lungs, which were evi-dent in his chest X-ray. Despite the 6 weeks of medical therapy persistent fever and recurrent emboli into lungs made the surgi-cal therapy as mandatory.

Patient was referred to our hospital for surgical therapy. Pul-monary arteriotomy was performed and the pulPul-monary valve was excised. There was a large pediculated vegetation over the cusps of the pulmonic valve (Fig. 1). Considering monocusp peri-cardial valve may improve the sudden hemodynamic change we have implanted a glutaraldehyde preserved pericardial patch to prevent further pulmonary valve insufficiency. Outlet VSD was closed with a pericardial patch. The patient was easily weaned from cardiopulmonary bypass. Pulmonary artery pressure (PAP) at the end of the operation was 50 mmHg. At postoperative 24th hour patient fell into the episode of pulmonary hypertensive cri-sis while he was still intubated. His PAP was 80 mmHg. Blood gas analysis showed marked hypoxemia and hypercapnia. He prog-ressed into the low cardiac output state. Despite the full medical therapy he eventually arrested. The autopsy showed a dilated right ventricle and several infarcts in the lungs.

In conclusion, excision of the pulmonary valve in the setting of pulmonary valve endocarditis is an acceptable option to pre-vent the septic emboli and to control the infection. However pul-monary valve insufficiency should be monitorized carefully and every effort should be made to prevent pulmonary hypertensive crisis in those patients.

Address for Correspondence: Tekin Y›ld›r›m, MD, Göztepe fiafak Hastanesi, Kalp ve Damar Cerrahisi Klini¤i, Fahrettin Kerim Gökay Cad. No:192,

Çemenzar, 34730, Göztepe, ‹stanbul, Tel: 0 216 565 44 44 / 11 57, Fax: 0 216 565 85 85, E-mail: ty@ttnet.net.tr

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