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Vibrating heart

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Vibrating heart

This is a surgical video of a patient with an ascending aortic aneurysm and severe aortic insufficiency (Fig. 1). On palpation of the heart, there was a significant thrill. When we recorded a video of the heart when the pericardium was opened, there was no visible abnormality (Video 1). We thought of taking a video in slow motion. On diastole, the heart was fibrillating (Video 2).

Bilgin Emrecan

Department of Cardiovascular Surgery, Faculty of Medicine, Pamukkale University; Denizli-Turkey

Video 1. Video of the beating of the heart of a patient with severe aortic insufficiency.

Video 2. Video of the beating of the heart in slow motion.

Address for Correspondence: Dr. Bilgin Emrecan, Gerzele Mahallesi 593/1 Sokak

No:42 Merkezefendi 20045 Denizli-Türkiye

Phone: +90 505 488 99 16 E-mail: bilginemrecan@yahoo.com

©Copyright 2018 by Turkish Society of Cardiology - Available online at www.anatoljcardiol.com

DOI:10.14744/AnatolJCardiol.2018.92597

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Figure 1. Surgical view of an ascending aortic aneurysm

Chronic thromboembolic pulmonary

hypertension versus fibrosing

mediastinitis

A 63-year-old woman with aggravating dyspnea was re-ferred to our center. Her symptoms began three months ago and worsened throughout this period. She reported a history of breast cancer treated with mastectomy and chemoradiation

Figure 1. (a) Diffusely decreased radiotracer uptake in the left lung along with multiple wedge-shaped pleural-based segmental pulmonary perfu-sion defects in both lungs. Planar images from different angles (POST, posterior; ANT, anterior) (b) Oblique coronal reconstructed view of CT pulmonary angiography (CTPA). Remarkable post-radiation right upper lobe pulmonary fibrosis (thin arrow) accompanied by extensive medias-tinal and bihilar fibrotic changes. Significant ostial stenosis of bilateral lower lobar arteries (thick arrows) as well as thromboemboli in left upper lobe segmental arterial branch (curved arrow). Complete occlusion of the right upper lobar arterial branch was noted. (c) Anterior–posterior view of pulmonary angiography. Significant ostial stenosis of the left lower lobe artery depicted in b (d) Oblique coronal reconstructed view of CTPA. Right side mastectomy and significant ostial stenosis of the right lower lobe pulmonary vein (RLL PV) (e) Coronal view of CTPA. Extensive bihilar post-radiation fibrosis with bilateral upper pulmonary vein occlusion (arrows)

a

b c

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E-page Original Images

Anatol J Cardiol 2019; 21: E-4-5

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therapy five years ago, with no recurrence noted in her sub-sequent follow-ups. She had also suffered from an episode of pulmonary emboli at the start of her chemotherapy. Echocar-diography revealed moderate right ventricular dysfunction and an elevated systolic pulmonary artery of 85 mm Hg as well as multiple perfusion defects in the ventilation-perfusion (V/Q) scan (Fig. 1a). Hence, the patient was referred to our center for further evaluation of chronic thromboembolic pulmonary hypertension (CTEPH). Pulmonary CT angiography (CTA) revealed extensive mediastinal and bihilar fibrotic changes, which diffusely en-croached pulmonary vasculature (Fig. 1b-e). Of note, co-existing small, pulmonary emboli were detected in few segmental arterial branches Figure 1b; these account for the perfusion defect noted in the initial V/Q scan.

Although fibrosing mediastinitis is primarily caused by granulomatous infections, it might be rarely observed in pa-tients with a history of radiotherapy (1). Diffusely decreased radiotracer uptake accompanied with perfusion defects in the V/Q scan might be inspected in both fibrosing mediastinitis and CTEPH. However, despite broad mediastinal and bilateral hilar fibrosis, almost normal peripheral branch arborization in CTA is more favorable for fibrosing mediastinitis than CTEPH. This discrimination plays a crucial role in patient management and future prognosis.

Reference

1. Morrone N, Gama e Silva Volpe VL, Dourado AM, Mitre F, Coletta EN. Bilateral pleural effusion due to mediastinal fibrosis induced by radiotherapy. Chest 1993; 104: 1276-8.

Kiara Rezaei Kalantari, Hadi Malek, Ahmad Amin, Jamal Moosavi*, Omid Shafe*,

Parham Sadeghipour*

Rajaie Cardiovascular Medical&Research Center,

*Cardiovascular Intervention Research Center, Iran University of Medical Sciences; Tehran-Iran

Address for Correspondence: Parham Sadeghipour, MD, Cardiovascular Intervention Research Center, Rajaie Cardiovascular

Medical&Research Center, Iran University of Medical Sciences, Vali-Asr Ave., Niyayesh Blvd., 1996911101 Tehran-Iran Phone: +989121454319

E-mail: psadeghipour@hotmail.com

©Copyright 2018 by Turkish Society of Cardiology - Available online at www.anatoljcardiol.com

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