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LETTER TO THE EDITOR

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Turkish Journal of Geriatrics 2011; 14 (4) 386

Volkan GENÇ

Ankara University, Faculty of Medicine Department of General Surgery ANKARA Tlf: 0312 508 26 76 e-posta: [email protected] Gelifl Tarihi: 01/04/2010 (Received) Kabul Tarihi: 17/05/2010 (Accepted) ‹letiflim (Correspondance)

Ankara University Faculty of Medicine Department of General Surgery ANKARA Volkan GENÇ1

Elvan Onur KIRIMKER1

fiiyar ERSÖZ1

Ahmet Serdar KARACA1

Erkin OROZAKUNOV1

Gökhan Ç‹PE1

L

ETTER TO THE

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DITOR

/E

D‹TÖRE

M

EKTUP

T

O

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HE

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DITOR

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terine prolapse is the herniation of uterus into or through the vagina. In UK, theannual incidence of hospital admission with prolapse is 20.4/10000. Confirmed risk factors are older age higher parity, vaginal delivery, race, family history, obesity and constipation (1). Menopause, long second stage of labor, musculoskeletal disea-se, trauma, smoking and increased intra-abdominal pressure are possible risk factors (2,3). Common complaints and symptoms are feeling of a vaginal bulge and pressu-re, urinary or fecal incontinence, feeling of incomplete voiding or defecation, weak or prolonged urinary stream, dyspareunia and lack of sexual sensation (4,5).

A 74-year-old woman who was treated due to esophageal variceal bleeding in gastroenterology service was referred to our surgical department for her recurrent umbilical hernia. She was diagnosed with chronic liver disease fifteen years ago and had six vaginal deliveries. On physical examination, recurrent umbilical hernia, massive ascites and uterovaginal prolapse (Figure 1) were detected. Prolapse deve-loped gradually during the last five years whereas formation of ascites had started 10 years ago. Operation for recurrent umbilical hernia and uterovaginal prolapse was proposed but she declined.

Multiparity causes recurrent levator ani injuries and high intraabdominal pres-sure certainly facilitates uterine prolapsed (6). Nevertheless ascites has the main ro-le in intraabdominal pressure increase. Umbilical hernia secondary to massive asci-tes is a common pathology with a high recurrence rate (7). We believe that this is the first case in English literature that demonstrates the association between chro-nic massive ascites and uterovaginal prolapse.

R

EFERENCES

1. Mant J, Painter R, Vessey M. Epidemiology of genital prolapse: observations from the Oxford family planning association study. Br J Obstet Gynaecol 1997;104:579-85. (PMID: 9166201).

2. Swift SE, Woodman P, O’Boyle A, et al. Pelvic organ support study (POSST): the dis-tribution, clinical definition and epidemiology of pelvic organ support defects. Am J Obstet Gynecol 2005;192:795-806. (PMID: 15746674).

3. Olsen A, Smith VJ, Bergstrom JO, Colling JC, Clark AL. Epidemiology of surgically managed pelvic organ prolapse and urinary incontinence. Obstet Gynecol 1997;89:501-6. (PMID: 9083302).

4. Doshani A, Teo REC, Mayne CJ, Tincello DG. Uterine prolapse. BMJ 2007;335:819-23. (PMID: 17947787).

5. Schaffer JI, Wai CY, Boreham MK. Etiology of pelvic organ prolapse. Clin Obstet Gynecol 2005;48:639-47. (PMID: 16012231).

6. Davis JD, Carroccio S. Massive uterovaginal prolapse in a young nulligravida with asci-tes: a case report. J Reprod Med 2007;52:727-9. (PMID: 17879835).

7. McKay A, Dixon E, Bathe O, Sutherland F.Umbilical hernia repair in the presence of cirrhosis and ascites: results of a survey and review of the literature. Hernia 2009;13:461-8. (PMID: 19652907).

Figure 1— Appearance of massive uterovaginal prolapse.

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