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Percutaneous treatment of the Gerbode defect causing heart failure after mitral valve surgery E-8

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Anatol J Cardiol 2020; 23: E-7-9 E-page Original Images

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Che-Yu Su, Chi-Wei Lee1, Chun-Yen Huang

Department of Emergency Medicine, Kaohsiung Medical University Hospital, Kaohsiung Medical University; Kaohsiung-Taiwan

1Institute of Medical Science and Technology, National Sun

Yat-Sen University; Kaohsiung-Taiwan

Address for Correspondence: Che-Yu Su, MD, Department of Emergency Medicine, Kaohsiung Medical University Hospital, Kaohsiung Medical University; No.100,

Tzyou 1st Road Kaohsiung 807, Kaohsiung-Taiwan

Phone: 886-7-3121101 ext: 7553 E-mail: money0967@gmail.com

©Copyright 2020 by Turkish Society of Cardiology - Available online at www.anatoljcardiol.com

DOI:10.14744/AnatolJCardiol.2019.11786

cardiography (TTE) revealed normal left ventricular diameters and ejection fraction, dilated left atrium, dilated right atrium (RA), and dilated right ventricle. Moderate tricuspid valve (TV) regurgitation was observed, and pulmonary artery systolic pressure was 65 mmHg. MV functions were within normal lim-its. However, a defect between left ventricle (LV) and RA, which

Percutaneous treatment of the Gerbode

defect causing heart failure after mitral

valve surgery

A 45-year-old female who underwent mechanical mitral valve (MV) replacement 10 years before presentation was hos-pitalized for decompensated heart failure. Transthoracic

echo-Figure 1. (a and b) Transthoracic echocardiographic images showing the Gerbode defect (GD) in the region marked with an arrow. (c) Image of the Amplatzer Duct Okluder-1 (ADO-1) in the postoperative GD region. (d) ADO-1 preventing the GD-related passage

a c b d a d b e c f

Figure 2. (a) Arrow indicates a connection between left ventricle and right atrium via the Gerbode defect. (b-d) Images of the Amplatzer Duct Okluder-1 (ADO-1) placement procedure. (e) Left ventriculography after releasing ADO-1. (f) Association of the mitral valve and ADO-1

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Anatol J Cardiol 2020; 23: E-7-9 E-page Original Images

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caused marked left-to-right shunting, was observed by TTE. The defect was located immediately above the septal leaflet of TV and below the anterior leaflet of MV, which was consistent with the characteristics of Gerbode defect (GD) (Fig. 1a and 1b, Video 1). Accordingly, left ventriculography was performed, and it re-vealed a shunt flow from LV to RA (oximetry shunt ratio=2). Ven-triculography revealed the following defect dimensions: 7 mm in length, 6.9 mm in width, and 10 mm from the left ventricular opening site (Fig. 2a). Due to a high risk of reoperation and the favorable anatomical location of the defect, its percutaneous closure with the Amplatzer Duct Occluder-I was performed (Fig. 2b-2d). After the procedure, no shunt flow was seen between LV and RA (Fig. 1c and 1d, Fig. 2e, Video 1). MV functions were found to be within normal limits (Fig. 2f).

Acquired LV–RA communication, also known as GD, after mitral valve surgery has been reported in the literature. Al-though the standard treatment for GD is surgical closure, pa-tients with a high risk of reoperation and a favorable anatomical

location of the defect can be treated by percutaneous closure procedures.

Informed consent: Informed consent was obtained from the patient. Video 1. Transthoracic echocardiographic images of the Gerbode defect before and after the procedure.

Emre Refik Altekin, Hüseyin Yılmaz

Department of Cardiology, Faculty of Medicine, Akdeniz University; Antalya-Turkey

Address for Correspondence: Dr. Emre Refik Altekin, Akdeniz Üniversitesi Tıp Fakültesi,

Kardiyoloji Anabilim Dalı,

Dumlupınar Bulvarı Akdeniz Üniversitesi Kampüsü, Konyaaltı 07070,

Antalya-Türkiye Phone: +90 242 269 68 04 E-mail: dremre29@yahoo.com

©Copyright 2020 by Turkish Society of Cardiology - Available online at www.anatoljcardiol.com

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